Chronic cerebral hypoperfusion accelerates amyloid β deposition in APPSwInd transgenic mice

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Chronic cerebral hypoperfusion accelerates amyloid beta deposition in APPSwInd transgenic mice.

Chronic cerebral ischemia may accelerate clinicopathological changes in Alzheimer's disease. We have examined whether chronic cerebral hypoperfusion accelerates amyloid beta deposition in amyloid protein precursor transgenic (APP-Tg) mouse. At 5, 8, and 11 months of age, C57Bl/6J male mice overexpressing a mutant form of the human APP bearing the both Swedish (K670N/M671L) and the Indiana (V717...

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Prominent Cerebral Amyloid Angiopathy in Transgenic Mice

From the Experimental Genetics Group,* Center for Human Genetics, Flemish Institute for Biotechnology, the Center for Transgene Technology and Gene Therapy, Center for Molecular and Vascular Biology, and Genetic Epidemiology, K.U.Leuven, Leuven, Belgium; the Janssen Research Foundation, Beerse, Belgium; Innogenetics, Gent, Belgium; Institut de Pharmacology Moléculaire et Cellulaire/CNRS, Valbon...

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The Influence of Chronic Cerebral Hypoperfusion on Cognitive Function and Amyloid β Metabolism in APP Overexpressing Mice

BACKGROUND AND PURPOSE Cognitive impairment resulting from cerebrovascular insufficiency has been termed vascular cognitive impairment, and is generally accepted to be distinct from Alzheimer's disease resulting from a neurodegenerative process. However, it is clear that this simple dichotomy may need revision in light of the apparent occurrence of several shared features between Alzheimer's di...

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ژورنال

عنوان ژورنال: Brain Research

سال: 2009

ISSN: 0006-8993

DOI: 10.1016/j.brainres.2009.07.078